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While GLP-1RAs can directly stimulate mTORC1 in some contexts, the net effect of the impaired mTOR signaling during sustained caloric deficit may favor catabolism [34]

The largest reductions were seen for lung and breast cancer: People with lung cancer who were on GLP-1s were 50% less likely to progress to stage 4 than those taking a DPP-4 inhibitor

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And so, that is another area where we need more data